Are Hairs in balding area different or is it solely from the environment?

cpio wrote:
There might be a test like rdkml mentioned where almost everybody test positive even if they dont have any symptoms. Such a test is completely worthless.

Wasn't trying to say they didn't have symptoms. It shows just how many ill people there are IMO.

CS's info is accurate IMO.

CausticSymmetry wrote:Interesting points anthonyspencer54. I remember that last I checked, sperm counts have plummeted about 30% or so among men. Probably all the foreign estrogens out there.

Nope, you are overstating it. Saying theyve plummeted 30% would be highly inaccurate. In some populations they have declined, in others they remain stable, in others they have increased. Blanket statements like the one you made simply can not be maintained.

http://www.harryfisch.com/pdf/Declining%20Worldwide%20Sperm%20Counts-Disproving%20a%20Myth.pdf

Sorry to interrupt, but isn't this about the 5th thread to appear in the last month about whether the MPB region has distinct androgen sensitivity and what role this might play in MPB?

It's a great topic. I'm just having a hard time keeping track of all the parallel threads...

It shows how wildly inaccurate that test is. It's an oxymoron to imagine people having some disease and then trying to prove it.

rdkml wrote:

cpio wrote:
There might be a test like rdkml mentioned where almost everybody test positive even if they dont have any symptoms. Such a test is completely worthless.

Wasn't trying to say they didn't have symptoms. It shows just how many ill people there are IMO.

CS's info is accurate IMO.

cpio wrote:
It shows how wildly inaccurate that test is. It's an oxymoron to imagine people having some disease and then trying to prove it.

Here is a quote on the Barnes Temerature Test which has been talked about on this forum for a number of years.

"Conventional lab testing -
When you have your blood work done to check your thyroid most physicians look at three types of thyroid hormone; TSH, T4 and free T3. Some physicians also check reverse T3/T4. All of these should be checked, however some physicians will only check one or two of these. Under diagnosed hypothyroidism is extremely common due to many people having either sub-clinical or functional hypothyroidism. Both of these conditions are not accurately detected using standardized serum blood testing. An older test that is called the Broda Barnes Basal body temperature is a test that you can perform yourself, at home. This was the test used for many years until a blood test was developed in the later part of the 20st century."

http://nourishholisticnutrition.com/thyroid/hypothryroidism-an-under-diagnosed-epidemic/

You just dont get it. But thats OK.

rdkml wrote:

cpio wrote:
It shows how wildly inaccurate that test is. It's an oxymoron to imagine people having some disease and then trying to prove it.

Here is a quote on the Barnes Temerature Test which has been talked about on this forum for a number of years.

"Conventional lab testing -
When you have your blood work done to check your thyroid most physicians look at three types of thyroid hormone; TSH, T4 and free T3. Some physicians also check reverse T3/T4. All of these should be checked, however some physicians will only check one or two of these. Under diagnosed hypothyroidism is extremely common due to many people having either sub-clinical or functional hypothyroidism. Both of these conditions are not accurately detected using standardized serum blood testing. An older test that is called the Broda Barnes Basal body temperature is a test that you can perform yourself, at home. This was the test used for many years until a blood test was developed in the later part of the 20st century."

http://nourishholisticnutrition.com/thyroid/hypothryroidism-an-under-diagnosed-epidemic/

LawOfThelema wrote:

CausticSymmetry wrote:Interesting points anthonyspencer54. I remember that last I checked, sperm counts have plummeted about 30% or so among men. Probably all the foreign estrogens out there.

Nope, you are overstating it. Saying theyve plummeted 30% would be highly inaccurate. In some populations they have declined, in others they remain stable, in others they have increased. Blanket statements like the one you made simply can not be maintained.

http://www.harryfisch.com/pdf/Declining%20Worldwide%20Sperm%20Counts-Disproving%20a%20Myth.pdf

Eh, I have no trouble believing sperm counts are plummeting, especially in 1st world countries just with simple observation.
Toxicity of the populace going up, vitamin/mineral deficiencies in every aspect increasing due to soil conditions, lack of sun, sedentary lifestyles, rapid increase of all diseases and immune disorders, rapid increase in obesity, depression, mental disorders etc, synthetic estrogens and bpa exposure poisoning the populace on a large scale, and testosterone levels dropping, thus sperm counts are also dropping. For some probably way more than 20%... imo 1+2=3

http://www.bmj.com/content/312/7029/467.full

http://www.ncbi.nlm.nih.gov/pmc/articles/PMC1470335/

http://www.andrologyjournal.org/cgi/content/abstract/27/4/560

CausticSymmetry wrote:At the risk of sounding like a broken record, I believe it all ties into thyroid dysfunction. Whatever the missing element is, whether it is a mineral, vitamin, presence of a toxin which disrupts the enzyme or receptor and ultimately disrupts thyroid hormone will negatively effect the androgen receptor. This I believe is intrinsically why a non-balding man is relatively unaffected by DHT vs a balding man.

In my own experience, at my worst level of health & hair, I had the following problems: bad breathe, poor diet, stress...my scalp was oily, gum health was poor.

Fast forward into the future - zero problems zero hair loss (+ more hair).

Cs, how bad was/ is your hairloss?

SlowMoe wrote:

CausticSymmetry wrote:At the risk of sounding like a broken record, I believe it all ties into thyroid dysfunction. Whatever the missing element is, whether it is a mineral, vitamin, presence of a toxin which disrupts the enzyme or receptor and ultimately disrupts thyroid hormone will negatively effect the androgen receptor. This I believe is intrinsically why a non-balding man is relatively unaffected by DHT vs a balding man.

In my own experience, at my worst level of health & hair, I had the following problems: bad breathe, poor diet, stress...my scalp was oily, gum health was poor.

Fast forward into the future - zero problems zero hair loss (+ more hair).

Cs, how bad was/ is your hairloss?

?

SlowMoe,

Have you been to the blog...I haven't been there in a while, but I remember him having pics up on there.

anthonyspencer54 wrote:SlowMoe,

Have you been to the blog...I haven't been there in a while, but I remember him having pics up on there.

Wasn't aware of one

Check out: immortalhair.org

Its CS's original blog. All of the science behind his regimen is there in the Theory and Physiology pages.

To chime in here,

AGA is largely not well-understood. What is clear is that DHT has genetic coding that it takes to the affected follicles and attachment unravels a sequence of reactions that results in miniaturization. Just like any genetic features, those follicles were programmed to miniaturize as a result of attachment and its subsequent mechanisms during embryonic stages when you were in womb. However, miniaturization does not happen to those who do not have AGA. This is a mystery of genetics as to why you have brown eyes and not blue and so on. It is more complicated here because hair follicle itself is structurally the same as the permanent ones in the same head and to non-aga follicles in other men-Why then miniturization in AGa-prone people! : I think once you discover this, you have the cure.

Furthermore, there was some experiment conducted where an aga-prone follicle was transplanted onto arms of the same subject. The prone follicle actually miniaturized eventually-Thus, it is the follicles that are at fault or I should say at disadvantage to the genetic coding, not the environment as far as the location itself is concerned. Of cpurse, dht binding starts a sequence which also includes immune response that starts choking the follicle.

Similarly, a follicle from arm was transplanted to the galea scalp region and it did not die-(hair transplants)

Also, know that dht message to aga prone follicles in galea is to start miniturizing and to eventually shut them down, whereas, dht message to your beard follicles is to grow hair, and god damn thicker and thicker with each shave.

AGA is a monster and cure is long overdue

My hair did not thin at all before I reached my thirties. The follicles had no problem
with DHT whatsoever for many years. So obviously my hair follicles are not inherently vulnerable to DHT because then they would have started to thin out in my teens when DHT was skyrocketing. Obviously there is another paramter involved and not just exposure to DHT (just like the JFK assassination - there had to be a second shooter). This "second shooter" has nothing to do with hormones, enzymes, digestion, thyroid, whatever, the second shooter is purely environmental - has to do with the galea, which why the balding follows a pattern.

See www.malepatternbaldness.net for diagrams.

The_Mentalist wrote:To chime in here,

AGA is largely not well-understood. What is clear is that DHT has genetic coding that it takes to the affected follicles and attachment unravels a sequence of reactions that results in miniaturization. Just like any genetic features, those follicles were programmed to miniaturize as a result of attachment and its subsequent mechanisms during embryonic stages when you were in womb. However, miniaturization does not happen to those who do not have AGA. This is a mystery of genetics as to why you have brown eyes and not blue and so on. It is more complicated here because hair follicle itself is structurally the same as the permanent ones in the same head and to non-aga follicles in other men-Why then miniturization in AGa-prone people! : I think once you discover this, you have the cure.

Furthermore, there was some experiment conducted where an aga-prone follicle was transplanted onto arms of the same subject. The prone follicle actually miniaturized eventually-Thus, it is the follicles that are at fault or I should say at disadvantage to the genetic coding, not the environment as far as the location itself is concerned. Of cpurse, dht binding starts a sequence which also includes immune response that starts choking the follicle.

Similarly, a follicle from arm was transplanted to the galea scalp region and it did not die-(hair transplants)

Also, know that dht message to aga prone follicles in galea is to start miniturizing and to eventually shut them down, whereas, dht message to your beard follicles is to grow hair, and god damn thicker and thicker with each shave.

AGA is a monster and cure is long overdue